Safety Reading Notes
Read safety context beside the research guide.
The TRPV3 source set should still be read with safety context in mind. Mechanistic or preclinical evidence should not be converted into consumer instructions, and product identity can change how closely a source applies. PMID 30697147
PubMed For Dummies Article
TRPV3 Evidence Review: the long-form source walk-through
- TRPV3 currently has 37 source-backed evidence row(s), so this page should be read as a research guide rather than a single conclusion. PMID 30697147
- The evidence classes most visible in the row language are mechanistic or pharmacological (20), and insufficient (17). PMID 33362478
- The study-design language most visible in the row language is Animal study (15), Narrative or expert review (12), Cellular or in vitro study (2), and other mapped categories (1). PMID 27498155
- The repeated topics are TRPV3 (12), TRP channel activity or ionotropic cannabinoid target mechanisms (11), TRPV4 (4), receptor, target, metabolic, or pharmacology mechanisms (3), and other mapped categories (7), which tells the reader where to start opening PubMed and DOI links. PMID 26698193
Start with the research question
TRPV3 is built from 37 source-backed evidence row(s) and 20 research source(s). The current evidence classes read as mechanistic or pharmacological (20), and insufficient (17), and the study-design language most often reads as Animal study (15), Narrative or expert review (12), Cellular or in vitro study (2), and other mapped categories (1). PMID 30697147
The row-level question is not simply whether TRPV3 is "good" or "bad." The useful question is what each row studied, what evidence class it received, and whether the source is close to the reader's actual question. The most repeated row topics are TRPV3 (12), TRP channel activity or ionotropic cannabinoid target mechanisms (11), TRPV4 (4), receptor, target, metabolic, or pharmacology mechanisms (3), and other mapped categories (7). PMID 33362478
Rows involving human participants, patients, or clinical source language. These rows are closer to everyday reader questions, but still depend on population, dose, route, comparator, and endpoint. PMID 33155670
Animal, cellular, or model-based rows. These can explain why a topic is being studied, but they should not be read as human-health instructions. PMID 33168643
Rows about receptors, enzymes, channels, metabolism, binding, signaling, or pharmacology. These explain plausibility without proving a consumer outcome. PMID 33629929
Rows where safety, tolerability, risk, product limits, or insufficient evidence need to stay visible next to the rest of the article. PMID 35483477
The lane labels are not a quality score. They are a reading method: keep human evidence, preclinical evidence, mechanisms, and uncertainty in separate mental boxes before deciding what a source can actually support. PMID 34259916
Where this page has the most source density
The largest bucket surfaced for this page is TRP channel activity or ionotropic cannabinoid target mechanisms. That does not automatically mean the topic is settled; it means this is where the current source trail is densest. The next visible bucket is TRPV3, which gives readers another way to see what the literature repeatedly circles. PMID 30697147
Source density should be read with evidence posture. A bucket can contain many rows and still be limited if the studies are indirect, mixed, preclinical, product-specific, or mostly review-level. The paragraphs below name the buckets directly and keep each explanation connected to a source record. PMID 33362478
Bucket chapters: what the literature is circling
TRP channel activity or ionotropic cannabinoid target mechanisms
TRPV3 appears in rows about TRP channel activity or ionotropic cannabinoid target mechanisms mechanisms. It currently draws from 6 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 30697147
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 30697147
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Evidence row 214
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mech... PMID 30697147
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Evidence row 364
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; study design: Animal study; outcome... PMID 21175579
TRPV3
TRPV3 appears in rows about TRPV3 mechanisms. It currently draws from 5 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 33362478
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 33362478
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Evidence row 1005
CBD modulates TRPV3; evidence class: insufficient (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 33362478
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Evidence row 1013
CBD modulates TRPV3; evidence class: mechanistic or pharmacological (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 40025806
Receptor, target, metabolic, and pharmacology mechanisms
TRPV3 appears in rows about Receptor, target, metabolic, and pharmacology mechanisms mechanisms. It currently draws from 3 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 27498155
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 27498155
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Evidence row 764
THCV modulates receptor, target, metabolic, or pharmacology mechanisms; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: receptor, target,... PMID 27498155
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Evidence row 780
THCV modulates receptor, target, metabolic, or pharmacology mechanisms; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: receptor, target,... PMID 21726418
TRP channel activity or ionotropic cannabinoid target mechanisms
TRPV3 appears in rows about TRP channel activity or ionotropic cannabinoid target mechanisms mechanisms. It currently draws from 3 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 33362478
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 33362478
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Evidence row 215
CBD modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 33362478
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Evidence row 361
CBD modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mech... PMID 35483477
TRPV3
TRPV3 appears in rows about TRPV3 mechanisms. It currently draws from 3 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 30697147
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 30697147
-
Evidence row 1004
THC modulates TRPV3; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 30697147
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Evidence row 1015
THC modulates TRPV3; evidence class: mechanistic or pharmacological (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 40141324
TRPV2
TRPV3 appears in rows about TRPV2 mechanisms. It currently draws from 2 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 37199723
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 37199723
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Evidence row 991
CBD modulates TRPV2; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV2 channel activity, binding, signaling, or pharmacology). PMID 37199723
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Evidence row 1001
CBD modulates TRPV2; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV2 channel activity, binding, signaling, or pharmacology). PMID 36747846
TRPV3
TRPV3 appears in rows about TRPV3 mechanisms. It currently draws from 2 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 20942817
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 20942817
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Evidence row 1011
Cannabinoids modulates TRPV3; evidence class: insufficient (population or model: Human participants or patients mentioned; study design: Narrative or expert review; outcome measure: TRPV3 channel activity, binding, signaling, d... PMID 20942817
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Evidence row 1014
Cannabinoids modulates TRPV3; evidence class: insufficient (population or model: Human participants or patients mentioned; study design: Systematic review; outcome measure: TRPV3 channel activity, binding, signaling, dermatolog... PMID 26845556
TRPV3
TRPV3 appears in rows about TRPV3 mechanisms. It currently draws from 2 research source(s), and mechanistic evidence should stay separate from human-outcome evidence. PMID 27498155
Read this bucket as mechanism or pharmacology context. Mechanisms can make the biology easier to understand, but they are not the same thing as a demonstrated effect in people. PMID 27498155
-
Evidence row 1006
THCV modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant me... PMID 27498155
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Evidence row 1007
THCV modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, o... PMID 38691614
Human evidence, mechanisms, and safety are different lanes
This page currently separates human evidence (0 row(s)), mechanistic evidence (20 row(s)), and safety/tolerability context (0 row(s)). That separation is the heart of the site. Mechanistic evidence can make a topic biologically interesting, but it should not silently become a human outcome. PMID 30697147
Human evidence still depends on population, dose, route, duration, product identity, and endpoint. Safety rows belong in the same reading path as benefit-oriented rows because formulation, co-exposures, prescription medications, impairment context, and higher-risk populations can change how close a source is to a reader's question. PMID 33362478
What this does and does not mean
- It means the page has a traceable source trail. It does not mean every bucket has the same clinical strength. PMID 18354058
- It means mechanisms, animal models, human studies, safety rows, and insufficient-evidence rows are being kept visible as separate evidence types. PMID 21175579
- It does not turn a preclinical mechanism into a consumer recommendation, and it does not treat one product, dose, route, or population as interchangeable with another. PMID 38691614
How to use the source table
The source-backed evidence table below is the audit trail. Each row keeps a public sentence connected to a source record when a PubMed ID or DOI is available. If a sentence feels important, the reader should be able to click through, inspect the study type, and decide whether the source is close to the question they care about. PMID 30697147
This is why the public page is intentionally layered. The top gives the reader a fast orientation. The bucket table groups repeated rows into readable topics. The article body explains the buckets using the actual evidence-row language. The source notes below walk through every evidence row before the source table repeats the technical trace. PMID 33362478
Source-reading checklist for TRPV3
- Open the linked PubMed or DOI record. PMID 21726418
- Check whether the source studied humans, animals, cells, chemistry, pharmacology, product testing, or a review of prior literature. PMID 37199723
- Compare the source product, dose, route, population, and endpoint to the question being asked. PMID 36747846
- Look for safety, tolerability, drug-interaction, impairment, pregnancy, pediatric, psychiatric, cardiovascular, and product-quality context before treating the bucket as settled. PMID 20942817
- Return to the evidence table when the article summary sounds too broad; the row is the audit unit. PMID 28945920
Source Notes
TRPV3 source-by-source reading notes
These notes pull every evidence row on this page into the readable article body before the source table repeats the audit trail. Each note keeps the row language beside the PubMed or DOI link when available.
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Evidence row 214
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels. -
Evidence row 215
CBD modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 33362478
Evidence class: insufficient. Source: An Analysis of the Putative CBD Binding Site in the Ionotropic Cannabinoid Receptors. -
Evidence row 216
THCV modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 27498155
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Pure Δ9-tetrahydrocannabivarin and a Cannabis sativa extract with high content in Δ9-tetrahydrocannabivarin inhibit nitrite production in murine peritoneal macrophages. -
Evidence row 357
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 26698193
Evidence class: insufficient; Study design: Narrative or expert review. Source: An Introduction to the Endogenous Cannabinoid System. -
Evidence row 358
Cannabinoids modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 33155670
Evidence class: insufficient; Study design: Narrative or expert review. Source: Temperature-sensitive transient receptor potential vanilloid channels: structural insights into ligand-dependent activation. -
Evidence row 359
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 33168643
Evidence class: insufficient; Study design: Narrative or expert review. Source: The Pharmacological Case for Cannabigerol. -
Evidence row 360
CBD modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; study design: Animal study; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 33629929
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol inhibits human glioma by induction of lethal mitophagy through activating TRPV4. -
Evidence row 361
CBD modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 35483477
Evidence class: insufficient; Study design: Narrative or expert review. Source: Pharmacological effects of cannabidiol by transient receptor potential channels. -
Evidence row 362
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (population or model: Cellular or in vitro model mentioned; study design: Narrative or expert review; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 34259916
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoids in the landscape of cancer. -
Evidence row 363
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: insufficient (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 18354058
Evidence class: insufficient; Study design: Animal study. Source: Plant-derived cannabinoids modulate the activity of transient receptor potential channels of ankyrin type-1 and melastatin type-8. -
Evidence row 364
THC modulates TRP channel activity or ionotropic cannabinoid target mechanisms; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; study design: Animal study; outcome measure: TRP channel activity or ionotropic cannabinoid target mechanisms). PMID 21175579
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Effects of cannabinoids and cannabinoid-enriched Cannabis extracts on TRP channels and endocannabinoid metabolic enzymes. -
Evidence row 764
THCV modulates receptor, target, metabolic, or pharmacology mechanisms; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: receptor, target, metabolic, or pharmacology mechanisms). PMID 27498155
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Pure Δ9-tetrahydrocannabivarin and a Cannabis sativa extract with high content in Δ9-tetrahydrocannabivarin inhibit nitrite production in murine peritoneal macrophages. -
Evidence row 778
THCV modulates receptor, target, metabolic, or pharmacology mechanisms; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; outcome measure: receptor, target, metabolic, or pharmacology mechanisms). PMID 38691614
Evidence class: mechanistic or pharmacological. Source: TRPV3 activation by different agonists accompanied by lipid dissociation from the vanilloid site. -
Evidence row 780
THCV modulates receptor, target, metabolic, or pharmacology mechanisms; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: receptor, target, metabolic, or pharmacology mechanisms). PMID 21726418
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabinoid actions at TRPV channels: effects on TRPV3 and TRPV4 and their potential relevance to gastrointestinal inflammation. -
Evidence row 784
THCV studied for Inflammation-related outcomes; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: inflammation-related or immune outcomes). PMID 27498155
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Pure Δ9-tetrahydrocannabivarin and a Cannabis sativa extract with high content in Δ9-tetrahydrocannabivarin inhibit nitrite production in murine peritoneal macrophages. -
Evidence row 971
THC modulates TRPV1; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPV1 channel activity, desensitization, binding, signaling, or pharmacology). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels. -
Evidence row 978
CBD modulates TRPV1; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV1 channel activity, desensitization, binding, signaling, or pharmacology). PMID 37199723
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol sensitizes TRPV2 channels to activation by 2-APB. -
Evidence row 989
THC modulates TRPV2; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPV2 channel activity, binding, signaling, or pharmacology). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels. -
Evidence row 991
CBD modulates TRPV2; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV2 channel activity, binding, signaling, or pharmacology). PMID 37199723
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol sensitizes TRPV2 channels to activation by 2-APB. -
Evidence row 1001
CBD modulates TRPV2; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV2 channel activity, binding, signaling, or pharmacology). PMID 36747846
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol sensitizes TRPV2 channels to activation by 2-APB. -
Evidence row 1004
THC modulates TRPV3; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels. -
Evidence row 1005
CBD modulates TRPV3; evidence class: insufficient (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 33362478
Evidence class: insufficient. Source: An Analysis of the Putative CBD Binding Site in the Ionotropic Cannabinoid Receptors. -
Evidence row 1006
THCV modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 27498155
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Pure Δ9-tetrahydrocannabivarin and a Cannabis sativa extract with high content in Δ9-tetrahydrocannabivarin inhibit nitrite production in murine peritoneal macrophages. -
Evidence row 1007
THCV modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 38691614
Evidence class: mechanistic or pharmacological. Source: TRPV3 activation by different agonists accompanied by lipid dissociation from the vanilloid site. -
Evidence row 1008
THC modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 21726418
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabinoid actions at TRPV channels: effects on TRPV3 and TRPV4 and their potential relevance to gastrointestinal inflammation. -
Evidence row 1009
CBD modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 37199723
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol sensitizes TRPV2 channels to activation by 2-APB. -
Evidence row 1010
CBD modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 36747846
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabidiol sensitizes TRPV2 channels to activation by 2-APB. -
Evidence row 1011
Cannabinoids modulates TRPV3; evidence class: insufficient (population or model: Human participants or patients mentioned; study design: Narrative or expert review; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 20942817
Evidence class: insufficient; Study design: Narrative or expert review. Source: Pharmacogenetics of new analgesics. -
Evidence row 1012
CBD modulates TRPV3; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; study design: Cellular or in vitro study; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 28945920
Evidence class: mechanistic or pharmacological; Study design: Cellular or in vitro study. Source: Human podocytes express functional thermosensitive TRPV channels. -
Evidence row 1013
CBD modulates TRPV3; evidence class: mechanistic or pharmacological (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 40025806
Evidence class: mechanistic or pharmacological. Source: Exploring Cannabidiol-TRPV3 Mediated Biological Activities-Findings From Molecular Docking, Simulation and Proteins Network Interactions. -
Evidence row 1014
Cannabinoids modulates TRPV3; evidence class: insufficient (population or model: Human participants or patients mentioned; study design: Systematic review; outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 26845556
Evidence class: insufficient; Study design: Systematic review. Source: A Systematic Review of Plant-Derived Natural Compounds for Anxiety Disorders. -
Evidence row 1015
THC modulates TRPV3; evidence class: mechanistic or pharmacological (outcome measure: TRPV3 channel activity, binding, signaling, dermatology-relevant mechanism, or pharmacology). PMID 40141324
Evidence class: mechanistic or pharmacological. Source: The Impact of a Quinone Scaffold on Thermo-TRPs Modulation by Dimethylheptyl Phytocannabinoids. -
Evidence row 1016
THC modulates TRPV4; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPV4 channel activity, binding, signaling, or pharmacology). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels. -
Evidence row 1017
CBD modulates TRPV4; evidence class: insufficient (outcome measure: TRPV4 channel activity, binding, signaling, or pharmacology). PMID 33362478
Evidence class: insufficient. Source: An Analysis of the Putative CBD Binding Site in the Ionotropic Cannabinoid Receptors. -
Evidence row 1019
THC modulates TRPV4; evidence class: mechanistic or pharmacological (population or model: Animal model mentioned; study design: Animal study; outcome measure: TRPV4 channel activity, binding, signaling, or pharmacology). PMID 21726418
Evidence class: mechanistic or pharmacological; Study design: Animal study. Source: Cannabinoid actions at TRPV channels: effects on TRPV3 and TRPV4 and their potential relevance to gastrointestinal inflammation. -
Evidence row 1021
CBD modulates TRPV4; evidence class: mechanistic or pharmacological (population or model: Human participants or patients mentioned; study design: Cellular or in vitro study; outcome measure: TRPV4 channel activity, binding, signaling, or pharmacology). PMID 28945920
Evidence class: mechanistic or pharmacological; Study design: Cellular or in vitro study. Source: Human podocytes express functional thermosensitive TRPV channels. -
Evidence row 1030
THC modulates TRPA1; evidence class: insufficient (study design: Narrative or expert review; outcome measure: TRPA1 channel activity, desensitization, binding, signaling, or pharmacology). PMID 30697147
Evidence class: insufficient; Study design: Narrative or expert review. Source: Cannabinoid Ligands Targeting TRP Channels.